Alzheimer's disease (AD) is a multifactorial neurological disorder with no current cure, creating a significant medical demand. Traditional theories attribute AD to the build-up of the toxic protein amyloid beta (Aβ) and the formation of neurofibrillary tangles of tau-protein. However, emerging evidence propose that impaired mitochondrial function and metabolic activity play a crucial role in AD's development and progression. Our laboratory's primary focus is to explore and uncover effective methods for preventing and treating Alzheimer's disease. To achieve this, we apply different cutting
